Swimming-Induced Pulmonary Edema: A Scoping Review and Analysis of Epidemiology, Pathophysiology, Diagnostics, Management, and Implications for Resource-Limited Care[1]
Summary
PRISMA-ScR scoping review of swimming-induced pulmonary edema (SIPE) with an emergency-medicine and resource-limited-care focus. SIPE = acute dyspnea, cough, sometimes hemoptysis during/after swimming, especially in cold water; pathophysiology is central blood pooling raising pulmonary capillary pressure, plus cold vasoconstriction and exercise-driven cardiac output. Risk factors: hypertension, female sex, cold-water exposure, TIGHT WETSUITS, prior SIPE. Lung ultrasound is a promising rapid diagnostic; management is supportive (rapid removal from water, oxygen, sometimes positive airway pressure); recurrence is significant.
So what
The prevention-and-recognition companion to the IPE severity cohort, and the source of the concrete water-event checklist: SIPE is a life-threatening but reversible condition in HEALTHY swimmers, its risk factors are partly modifiable at the event (wetsuit fit, cold-water acclimatization, prior-episode screening), lung ultrasound belongs in the swim-finish medical kit, and the first move is get-them-out-and-oxygen. Underrecognition is the named danger - which is exactly why a curated MGMI page matters. Together with Kociuba, Di Masi, and Windsor it makes a four-entry water-events shelf.
Key findings
- Scoping review of SIPE for emergency/prehospital and resource-limited settings
- Risk factors: hypertension, female sex, cold water, tight wetsuits, prior SIPE
- Pathophysiology: central pooling + cold vasoconstriction + exercise cardiac output
- Lung ultrasound a promising rapid diagnostic; management supportive (remove/O2/PAP)
- Significant recurrence; underrecognition the named hazard
Read the paper
- Publisher (DOI) ↗may be paywalled
- PubMed ↗abstract · free